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Mitochondrial-mediated ROS cause oxidative damage to cellular structures, particularly mitochondrial DNA, somatic mutations, genome instability, inactivation of tumor suppressor genes, alterations in metabolic and signaling pathways with simultaneous activation of compensatory antioxidant mechanisms, all of which contribute to cell transformation and thus promote various types of cancer [42]
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Some participants also saw their symptoms improve to the point where they were considered mild or no longer clinically significant after one year of treatment
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In practice, both options can be effective, success largely depends on consistency, proper dosing, and accompanying lifestyle adjustments such as balanced meals and physical activity
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