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Co-administration of liraglutide 1.8 mg (at steady state) and insulin detemir (single dose) in patients with type 2 diabetes produced an additive glucose-lowering effect without affecting the pharmacokinetic profile of either agent (as evaluated by AUC, C max and t max ) [56]

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This group also studied mice with doxorubicin-induced cardiomyopathy and found that ferroptosis played an important role, possibly because doxorubicin upregulates HO-1, releasing free iron and leading to the generation of oxidized lipids in the mitochondrial membrane