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Kluwer Academic Publishers, Dordrecht, ISBN 1-4020-0178-9

COPA mutations impair ER-Golgi transport and cause hereditary autoimmune-mediated lung disease and arthritis

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In light of these intracellular observations (Kokame et al, 1996), we hypothesize that changes in the intracellular redox state associated with elevated homocysteine levels can alter the expression of multiple genes involved in other specific cellular functions that could be partially responsible for vascular disorders associated with moderate hyperhomocysteinemia