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In fact, these mice had substantially increased fungal burdens and enhanced fungal dissemination due to impaired phagocytosis, antigen presentation, and cytokine/ROS production of their monocytes ( Alcaligenes faecalis for our in vitro experiments while the RNA-seq datasets were generated using heat killed Candida albicans ( Alcaligenes faecalis ), zymosan ( fungi ), and yeast-derived BDG induce a similar pro-inflammatory phenotype in primary human monocyte-derived macrophages and primary human monocytes (42, 43)

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