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We find that, although GSH cannot protect against pink-1 or pdr-1 loss-of-function in the context of -synuclein alone, the joint additive neurodegenerative phenotype of both pink-1 and pdr-1 was partially rescued by GSH (Figure 4f) to a degenerative state reminiscent of loss of pink-1 or pdr-1 alone

Habous, M

Meanwhile, the upregulation of MDM2 suggests that the p53 signaling pathway may be inhibited, which to a certain extent reduces the ability of cells to repair DNA damage and may prompt cells to evade apoptotic mechanisms, further accelerating tumor progression

Autophagy and tau protein