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In the body, it is broken down into L-cysteine and then further metabolized by connecting it to glutamate and glycine to form glutathione
2019) An example of FUS at the end-organs, such as the spleen in case of inflammatory diseases or pulmonary arterial hypertension (Ahmed et al
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Previous studies have demonstrated that gut-derived GLP-1 activates vagal sensory nerves, triggering insulin secretion via brain-autonomic nervous system pathways, a mechanism referred to as the "neuro-incretin effect" [51, 59]
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