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Carnitine acetyltransferase (CrAT) can act as safety valve to catalyze the reversible transfer of acetyl groups between acetyl-CoA and acetylcarnitine, promoting the combination of excess acyl groups with carnitine, increasing the membrane permeability of these products and thus removing them from the mitochondria, allowing acylcarnitine to be formed in the mitochondrial matrix and exported to the plasma [7]
No significant differences were observed with respect to the percentage of lung lesions when the entire study population of cohort 2 was analyzed
It is unclear whether the effects on weight are sufficient, by themselves, to enable fibrosis regression
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