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As individuals age, the bidirectional communication between muscle and vascular systems becomes compromised, resulting in a reduction of capillary density, a decrease in the bioavailability of endothelial nitric oxide (NO), and an overall decline in muscle perfusionthese elements collectively contribute to the intensification of anabolic resistance and the acceleration of sarcopenic progression (5, 6)

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For example, mutations in the p53 tumor suppressor gene, often caused by oxidative stress, are frequently observed in various cancers [99]

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In some cases, it can exert pro-oxidant effects, potentially worsening oxidative stress