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The impact of defective GLP1R cell surface expression suggests that pharmacological chaperons, molecules known to facilitate proper export of proteins to the cell surface, might be of therapeutic relevance for mutation carriers, as already shown for other important metabolic receptors such as melanocortin MC4 receptor

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This effect is compounded by the fact that fat loss is often disproportionate, with the face being one of the first areas to show volume depletion

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More recently, the probiotic Lactobacillus plantarum SG5 has been shown to ameliorate motor deficits, neuronal death and the decreased colonic GLP-1 created by MPPT, indicating that the neuroprotective effect of SG5 likely involves modulation of the gut microbiota and, significantly, the GLP-1/PCG 1 pathway [49]