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Activation of the reverse transsulfuration pathway through NRF2/CBS confers erastin-induced ferroptosis resistance
TDP-43 undergoes mislocalization to the cytoplasm of neurons and glial cells, forming insoluble aggregates and inclusion bodies, which contribute to TDP-43 proteinopathies such as amyotrophic lateral sclerosis and frontotemporal lobar degeneration [101, 102]
It also promotes the promotion of nitric oxide (NO)
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