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Unlike many therapeutic peptides, Dihexa can cross the blood-brain barrier and promote BDNF signaling , a key process for: These effects have made Dihexa of great interest in treating neurological diseases, age-related cognitive decline, and more recently, brain fog and fatigue

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Animals sacrificed 5 days after transient MCAO and treatment with salvinorin A demonstrated increased density of microglia near the infarction in the cortex and corpus striatum but with fewer microglia expressing CD16, a marker for pro-inflammatory microglia/macrophages in the cortex (Misilimu et al., 2022)

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