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This upregulates genes involved in fatty-acid uptake/oxidation, OXPHOS, the TCA cycle, and mitochondrial biogenesis, shifting substrate use toward lipids and increasing energy expenditure
Methylcobalamin should not be used in patients with early hereditary optic nerve atrophy (Lebers disease)
Blocking NNMT raises cellular NAD+, which in research supports fat-cell metabolism and fat loss
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Presented for research literature review only Cifuentes ME et al
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