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Treatment of NRAS-Q61L mutated AML cells with the ferroptosis-inducing molecule erastin resulted in enhanced ROS levels and cytosolic translocation of HMGB1 leading to cell death, whereas this effect was not seen in unmutated cell lines (96)

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In A-induced AD model mice and APP23/PS45 double transgenic AD model mice, UA treatment promotes A clearance and improves cognitive deficits by restoring TFEB-dependent autophagy [217](Fig