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Differential contributions of C3, C5, and decay-accelerating factor to ethanol-induced fatty liver in mice

Peroxidase catalytic cycle of MCM-41-entrapped microperoxidase-11 as a mechanism for phenol oxidation

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It should be noted that we do not include mitochondrial glutathione, which the Jaeschke group has found to be fundamental in APAP pathophysiology [36]

Hence, a peptide was designed by substituting the negatively charged residues D42, D48, D49, E51, E56, and D57 within the p53 TAD PEP with alanine to increase its binding affinity to FOXO4 FHD and enhance membrane penetration