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It emphasizes that mild or transient heat stress primarily activates protective proteostasis-maintaining responses, whereas sustained or severe heat stress may exceed the compensatory capacity of the molecular chaperone system, thus promoting oxidative stress, apoptosis, and impaired spermatogenesis

Toxicol Mech Methods (2003) 13(2):837

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Necroptosis plays a dual role in tumour immune regulation, as it can activate antigen-specific immune responses by releasing damage-associated molecular patterns, while RIPK3/MLKL-mediated IL-1 release can suppress T-cell function and promote tumour immune escape [59]