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Current cagrilintide peptide research focuses on its interaction with receptor-mediated signaling pathways and peptide-driven molecular mechanisms at the cellular level
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Viewed from this perspective, mTORC1 is a pro-oncogenic factor as it represses autophagy by either directly inhibiting multiple stages of autophagy or indirectly regulating lysosomal biogenesis (Rabanal-Ruiz et al., 2017) (Figure 2)
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