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Their primary mechanisms include: Enhancing insulin secretion from pancreatic beta cells in a glucose-dependent manner Suppressing glucagon release , which reduces hepatic glucose production Slowing gastric emptying , promoting satiety and reducing appetite Acting on central nervous system pathways involved in appetite regulation These combined effects lead to improved glycaemic control in people with type 2 diabetes and significant weight reduction in those with obesity
2 Further research has also demonstrated that GST is expressed abundantly in tumor cells and associated closely to carcinogenesis, tumor formation, and chemotherapy resistance
Epithelial thickness was analyzed with the QuPath analysis software (version 0.5.1)
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