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Transient exposure to hydrogen peroxide causes an increase in mitochondria-derived superoxide as a result of sustained alteration in L-type Ca 2+ channel function in the absence of apoptosis in ventricular myocytes

NMDA-R-ionophores are reported to be regulated by endogenous glutathione (antioxidant) and slightly activate the glutamate and NMDA-Rs induced calcium influx [43, 44]

Phase 1b study of pegylated arginine deiminase (ADI-PEG 20) plus pembrolizumab in advanced solid cancers

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