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The article describes semaglutide binding GLP-1 receptors in the arcuate nucleus and lateral hypothalamus, where it suppresses neuropeptide Y and agouti-related peptide, two neurochemicals that drive hunger, while increasing activity of pro-opiomelanocortin neurons that signal fullness
The dual-chamber device may further include a drug discharge device, which includes an actuatable piston rod to apply a driving force to the second elastomer plug
These changes reduce calculated cardiovascular risk substantially
Early studies and reviews suggest GLP-1 medications may reduce binge episodes in some individuals by reducing cravings and reward-driven eating
G.et al (2023)
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