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Elsevier, Amsterdam

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This bidirectional interaction enables restoration of normal NO balance critical for proper sphincter timing

Key ways it appears to mimic or amplify natural gastrointestinal and tissue repair mechanisms observed in animal studies: Strongly upregulates angiogenesis through multiple pathways (VEGF, EGR-1, FAK-paxillin) in injured tissues, similar to the bodys own response during normal wound healing and tendon/ligament repair

When it binds to copper ions, it forms the GHK-Cu complex, which plays a role in wound healing, collagen synthesis, and skin repair