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Treatment of human and mouse small intestine organoid models with SCFAs increases the number of GLP-1-producing cells twofold 284 , and SCFA infusion activates colonic EECs to increase GLP-1 release in mice 281
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Although the primary impact of Semaglutide is on glucose metabolism and appetite regulation through GLP-1 receptor pathways, there exists an indirect possibility for these mechanisms to affect blood flow
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