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When exenatide received FDA approval in 2005, GLP-1R was understood primarily as a pancreatic target, and the incretin system looked like a clean metabolic story

While the mechanism by which GSTO1 interacts with these genes and pathways remains to be fully characterized, GSTO1 catalytic activity appears to be important for this regulation

Our real-world evidence of the comparative effectiveness of GLP-1RAs versus insulin complements existing trial evidence by translating the efficacy of GLP-1RAs in clinical trials to its effectiveness in clinical practice among a broader spectrum of real-world patient populations for supporting the rational selection of GLAs in clinical practice [48]

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