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One plausible explanation for these results is that the gpoA mutant is unable to effectively counteract the elevated ROS concentrations encountered within the intracellular environment of host cells, despite the partial compensatory activity of the TpxD and AhpD peroxidases (Fig
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The mechanism may be indirect, but it is consistently proven in large-scale clinical trials
In particular, the pharmaceutical sector has shown a growing interest in using L-carnitine for nutritional therapy
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