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doi: 10.3945/ajcn.2009.28449C

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The single-cell transcriptomic analysis of GA patients revealed that the encoding gene, HIF-1A, is significantly upregulated in the monocytes of GA patients ( FAs can increase the de novo synthesis of HIF-1 at the translational level through fatty acid-binding protein 5 (FABP5) ( FAs may also increase uric acid levels by inhibiting the excretion process

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When the right peptide binds with the right receptor, it triggers a specific function, such as releasing hormones, burning fat, or increasing metabolism