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Additionally, GLP-1 activates related intracellular signaling pathways, promoting -cell regeneration, inhibiting -cell apoptosis, and improving -cell function, thereby lowering blood glucose levels, with its effects being glucose concentration-dependent (58, 59)
The authors found no significant increase in the LLA for SGLT2 inhibitors compared to GLP-1 RAs, while they were associated with a lower risk compared to dipeptidyl peptidase-4 (DPP-4) inhibitors, but a higher risk compared to sulphonylureas [99]
The most clinically recognised cause is a temporary drop in potassium as your body rapidly makes new red blood cells
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