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The proposed mechanism involves TB-500s regulation of the actin cytoskeleton during the remodeling phase of wound healing, where the balance between myofibroblast contraction (which drives scar formation) and normal cell migration and differentiation (which drives regeneration) determines tissue outcome
New England Journal of Medicine, 387(24), 22452257
These approaches enable investigation of GSH/GSSG redox systems, glutathione-associated enzyme activity, thiol chemistry, and redox-regulated biochemical processes
Nogueira, V
10.7326/ANNALS-24-01249 19 GibbsA
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