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Using advanced tracer methodologies, the researchers identified a fascinating metabolic phenomenon they termed "brain glucose steal." Due to systemic mitochondrial impairment, non-brain organs were essentially stealing glucose intended for the central nervous system, starving the brain of energy and driving cognitive dysfunction

Characterizing the mechanism of action for mRNA therapeutics for the treatment of propionic acidemia, methylmalonic acidemia, and phenylketonuria

Hong Y, Zou H, Hu Y, Fei F, Liang L, Liu D, et al

In the present study, supplementation of withanolide A reversed hypoxia induced down regulation of Nrf2 in the hippocampal region of the brain with concomitant increased expression of GCLC

GHK-Cu is a tripeptide glycyl-L-histidyl-L-lysine complexed with a copper(II) ion