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Biomechanical forces in atherosclerosis-resistant vascular regions regulate endothelial redox balance via phosphoinositol 3-kinase/akt-dependent activation of Nrf2

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In one rat study, BPC-157 administration promoted tendon fibroblast outgrowth, cell survival under stress, and cell migration through the FAK-paxillin signaling pathway

Oscillating glucose (OsG), leading to glycemia fluctuation every 6 h for 24 h, is more deleterious to endothelial function and oxidative stress than stable glucose in normal and T2DM patients 6

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