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Kla modification activates the promoter region of the PD-L1 gene, and this epigenetic regulation provides a molecular basis for high PD-L1 expression, further inhibiting the antitumor activity of CD8 + T cells (94, 95)
Conversely, higher folate and cobalamin statuses are associated with greater percentage rises in postload homocysteine after adjustments for age and sex (Silberberg et al, 1997)
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