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Together with its oxidized partner (GSSG), GSH maintains cellular redox homeostasis 1 , regulates protein functions through S -glutathionylation 2 , contributes to iron-sulfur cluster maturation 3 , and acts as a signalling molecule to directly activate gene expression 4,5

The expression of exhaustion markers did not correlate with cytotoxic markers, indicating they are not upregulated by cytotoxic cell activities

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