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This finding is particularly significant, as CAP is known to bind to the transmembrane domains of TRPV1 channels 60 , suggesting that exendin 939 is a selective and potent antagonist for pain relief without competitively displacing CAP at its binding site
We used concentrations within the range reported in alcohol-dependent individuals (~50125 mM) and maintained exposure for 24 h in microglial and neuronal cell cultures, consistent with previous studies [22, 31, 32]
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