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How peripherally administered GLP-1 induces satiety mechanistically is unclear, but it is postulated that signals being generated by the binding of GLP-1 to its receptor on neurons in the GIT, hepatoportal bed and CNS mediate this effect

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In contrast, patients with slow metabolism experience appetite suppression with semaglutide for longer

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Notably, the docking orientation of the top-ranked peptide was observed to be different compared to that of the GLP-1