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Follow up studies with in vivo [ 13 C 6 ]-glucose tracing on liver and kidney 24 h after AKI revealed 4 major findings: (1) increased flux through glycolysis and the tricarboxylic (TCA) cycle in both kidney and liver

Although clinical differences between selenium treatment and placebo could not be observed in any subgroup, further studies may build on these findings to define a specific responder phenotype that may be susceptible to anti-inflammatory treatment strategies with high-dose selenium

An analysis conducted by Zller et al.[61] using mice and exosomes demonstrated that repeated administration of exosomes derived from myeloid-derived suppressor cells (MDSCs) obtained from healthy donors prevented disease progression and promoted regional hair regrowth in the C3H/HeJ AA mouse model [61]

10.1016/j.ejphar.2012.01.010 Summary Keywords acetaminophen, phytic acid, iron, SIRT -1, CYP2E1 , liver Citation Hassan HM, Abdel-Halim NHM, El-Shenbaby I, Helmy MA, Hammad MO, Habotta OA, El Nashar EM, Alghamdi MA, Aldahhan RA, Al-Khater KM, Almohaywi B and Farrag EAE (2024) Phytic acid attenuates acetaminophen-induced hepatotoxicity via modulating iron-mediated oxidative stress and SIRT-1 expression in mice

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