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Indeed, gut dysbiosis, especially related to obesity of high-fat diet, can increase EC activity, thus, determining an increased gut permeability and consequent LPS translocation
Studies in animal models of neurological damage document not only preservation of existing neurons but recovery of function suggesting actual repair and reconnection of damaged neural circuits through enhanced synaptogenesis and neurite outgrowth
In a 26-week rat oral toxicity study, AOD-9604 produced dose-dependent changes in serum osteocalcin (a bone-turnover marker) at week 13, with the direction reversing by week 26
Supports glutathione pathways
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