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On August 8th, the company had a press release to announce that the primary endpointcardiovascular death, heart attack or strokewas reduced by 20%
This slow, serotonin-based mechanism is a core pharmacological reason fluoxetine carries no recognized abuse potential
[1] [2] It should be noted that the precise dosing regimen and titration schedule are still under investigation and may differ from those used in trials if the medicine is eventually approved
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