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Mechanism of Action: IGF-1 LR3 vs IGF-1 DES Both IGF-1 LR3 and IGF-1 DES bind to the IGF-1 receptor , triggering powerful anabolic pathwaysprimarily PI3K/Akt/mTOR for muscle protein synthesis and MAPK for cell proliferation
Presented for research literature review only Velasco et al
GCL comprises a catalytic (GCLC) and a modifier (GCLM) subunit, and its activity can be readily inhibited with DL-buthionine-sulphoximine (BSO) [13]
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