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Focusing on the MMP-9/TIMP-1 axis, which is a pivotal molecular pathway governing airway remodeling and fibrosis, the study demonstrated that in a cigarette smoke plus LPS-induced rat model of COPD, the expression of MMP-9 in lung was markedly upregulated and the regulation of TIMP-1 was imbalanced, resulting in the coexistence of excessive extracellular matrix degradation and aberrant deposition, thereby driving airway wall thickening and fibrotic progression

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The increased FAO driven by carnitine leads to an increase in intracellular acetyl-CoA levels, which can be transferred to the nucleus for acetylation of histone H3K27, thereby activating GATA3 gene expression and promoting the differentiation of triple-negative breast cancer cells to the luminal cell state [36]