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In the nigrostriatal tract system, dopamine undergoes autooxidation or oxidative deamination by monoamine oxidase-B, both of which aggravates with age, leading to increased production of peroxides and hydroxyl radicals 41
The two pathways converge on somatotroph GH release through independent second-messenger cascades, and the combined-receptor pharmacology in healthy-adult human preparations across the Bowers laboratory work in the 1980s and 1990s reported a larger GH excursion under combined GHRH-analog plus GHRP-class administration than under either compound singly [6]
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