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In parallel, oxidative stress contributes to pathogenesis through an imbalance between pro-oxidant and antioxidant systems, promoting cellular injury and immune dysfunction
Oxidative stress also causes breaks in the DNA strand, which leads to changes in gene expression and genetic mutations (8, 63)
Konopka et al., 2020), cytoplasmic mislocalization of nuclear proteins (Neumann et al., 2006), mitochondrial dysfunction (Albers and Beal, 1997), proteasomal and autophagic dysfunction (Chen et al., 2012), ER stress (Nagata et al., 2007
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