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Beyond mitochondrial dysfunction, the association between apoptosis and endoplasmic reticulum (ER) stress has also been widely studied (37, 83, 110)
We therefore postulated that IGF-1R mAbs could represent a viable therapeutic tool to target IGF-1 action, and potentially mimic the beneficial effects associated with diminished IGF-1 signaling observed in animal models
Protein damage tryptophan fluorescence (decrease in OD)
Furthermore, some medications may interfere with glutathione levels, so its crucial to inform our healthcare practitioners about any medications you are currently taking
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