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However, excessive TGF-1 signalling can worsen dopaminergic deficits in MPTP models, underscoring the context-dependent role of TGF-1 in PD [207, 209]
The easiest way to understand how this works is to envision the IGF-1 receptor as a lock and IGF-1 LR3 as the key
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Patients should not add CJC-1295, ipamorelin, AOD-9604, BPC-157, TB-500, or another peptide to an existing treatment without medical guidance
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