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Additionally, the forebrain lateral septum (LS) appears to mediate the non-avoidant food intake reducing effects of GLP-1Rs in that direct exenatide injection into the rat LS suppresses chow intake but does not enhance kaolin intake (85)
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[6] [10] The correct tier is therefore investigational, development halted for lack of demonstrated efficacy not approved, and emphatically not a compound that succeeded in trials
They don't replace the deficit itself
Not every restrictive eating disorder has the same cause, Huckins warns, and a GLP-1 could further mess with the disrupted circuitry that often drives it
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