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Larson AM, Polson J, Fontana RJ
Normalized data was then statistically analyzed to evaluate differences across experimental groups
Beyond neurological implications, the research extends to oncology, particularly tumors harboring mutations in the KEAP1 gene
This vulnerability is compounded by an age-dependent decline in endogenous antioxidant defenses, contributing significantly to neuronal dysfunction and neurodegenerative pathogenesis
After autophagosome formation, SNARE proteins such as VAMP8, STX17, and SNAP29 facilitate the fusion of autophagosomes with lysosomal membranes to form autolysosomes (Itakura et al., 2012)
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