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GIP receptor has been found to be expressed in the central nervous system

However, recent comprehensive studies are challenging this belief and providing clarity for both patients and healthcare providers

(2008) described the discovery and characterization of the tissue-protective EPO receptor (now called the innate repair receptor) and demonstrated that engineered EPO derivatives lacking erythropoietic activity retained cytoprotective signaling, laying the foundation for ARA-290 development [2]

"The biggest concern is damage to the cornea, the clear part of the front of the eye

This is the common administration method for studying musculoskeletal healing in animals [4]