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Moreover, high Glp1r expressing and T cells can further decrease GLP-1 concentration in media from GLUTag cells in the presence of exendin-4, suggesting that Glp1r- expressing and T cells act as a sink for local GLP-1 production (He et al., 2019)
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Assessing the requirement of CD36 for GLP-2-mediated increases in intestinal-TG secretion are complicated by the increased fatty acid absorption (as shown by appearance of radiolabel in plasma), TRL-TG and TRL-apoB48 secreted by Cd36 / mice compared to wild-type controls (Hsieh et al., 2009)
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