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Deficiency of vitamin B12 and its relation with neurological disorders: A review
GLP-1 agonists work primarily through appetite suppression and slower gastric emptying
Two downstream consequences are mechanistically central: nicotinamide is preserved for re-entry into the NAD+ salvage pathway (raising intracellular NAD+ availability), and SAM is preserved for other methylation reactions
In the absence of coenzyme B12, tetrahydrofolate cannot be regenerated from its inactive storage form, 5-methyl tetrahydrofolate, resulting in functional folate deficiency
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