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Patients with Long COVID, ME/CFS, and dysautonomia often suffer from gastrointestinal malabsorption, genetic methylation defects (like MTHFR mutations), and impaired liver function

Lets break down some possible reasons people feel different on GLP-1 meds: 1

This physiological state, diabetic ketoacidosis (DKA), results from a reduced supply of glucose, due to lack of circulating insulin in the absence of therapeutic injection, and a concomitant increase in fatty acid oxidation due, in large part, to an unregulated increase in circulating glucagon

Some people leave with a BPC-157 prescription

More recently, dual and triple agonists targeting additional incretin and metabolic hormone receptors have demonstrated enhanced therapeutic potential, revolutionizing the landscape of MASLD treatment [11,12]