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It is well assessed that the CAC catalyzes the entry of acylcarnitines into the mitochondrial matrix in exchange for free carnitine (Figure 3 and Table 1)
The exception is an increase of the mitochondrial 5,10-me + -THF, likely due to an upregulation of the mitochondrial 1 C metabolism in galactose 32
Beuckmann CT, Fujimori K, Urade Y, Hayaishi O
Its molecular structure enables strong copper chelation, while its physicochemical properties require careful handling to preserve complex stability in laboratory settings
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