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These results were further supported by another study from the same lab, whereby GLP-1 was shown to stimulate myocardial glucose uptake via mechanisms that were p38 MAP kinase and NO dependent, but independent of adenylyl cyclase or Akt [73]
Yang, Y., Mohammed, F
Enhanced colonization of epithelial-adherent pathogens such as AIEC exacerbates mucosal permeability, reshapes microbial community composition, and initiates inflammatory cascades via upregulation of pro-inflammatory genes, ultimately driving intestinal inflammation (81)
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