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This may involve targeting the CBX3/NRF2/GPX2 axis with CBX3 or NRF2 inhibitors or impairing antioxidant buffering systems such as glutathione synthesis (e.g., via GCLC suppression)
This new protocol was confirmed by performing the BlandAltman plot analysis of Gpx activity in matched samples through the GpxDTNB assay
IL-1 is a typical inflammatory mediator produced by nucleotide-binding domain, leucine-rich-containing family, pyrin domain-containing 3 (NLRP3) inflammasome, leading to myocardial hypertrophy, heart failure, and coronary atherosclerosis [85, 86]
Glutathione reductase modulates endogenous oxidative stress and affects growth and virulence in Avibacterium paragallinarum
Oxidative stress is involved in endothelial damage resulting from endothelial malfunction, which increases endothelial permeability and inflammatory cell infiltration in the arterial wall [69]
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